POST-PRANDIAL DYSMETABOLISM AND THE VASCULAR COGNITIVE DECLINE

This is an automatically generated default intro template – please do not edit.

	Read full article
Article Title:	POST-PRANDIAL DYSMETABOLISM AND THE VASCULAR COGNITIVE DECLINE
Authors:	Corina ZORILĂ
Affiliation:	“Vasile Goldis” Western University Arad, România
Abstract:	Preventing or postponing the onset of cognitive decline and delaying or slowing its progression would lead to a consequent improvement of health status and quality of life in older age. The diet is considered an important factor of a healthy state but there are debates about what the diet must to cover. Dietary and lifestyle factors play also a central role in the etiology of post-prandial dysmetabolism. Since several dietary factors affect the risk of cardiovascular disease, it can be assumed that they also influence the risk of dementia .Promising non-pharmacologic approaches to the normalization of post-prandial dysmetabolism are evolving. The traditional Mediterranean diet, which is rich in minimally processed natural foods, low in caloric density but high in nutrient density, have been associated with improved CV health and even with the limitation of cognitive decline. We examine the possible role of macronutrients and food nutrients, with a particular focus on the Mediterranean diet in modulating the risk of Alzheimer disease and dementia.
Keywords:	dementia, Mediterranean diet, oxidative stress
References:	Panza F, Solfrizzi V, Colacicco AM et al. Mediterranean diet and cognitive decline. Public Health Nutr. 7(7), 2004, 959–963. Peters R. The prevention of dementia. Int. J. Geriatr. Psychiatry 24(5), 2009, 452–458 The World Alzheimer Report 2011 Anstey KJ, Lipnicki DM, Low LF. Cholesterol as a risk factor for dementia and cognitive decline: a systematic review of prospective studies with meta-analysis. Am. J. Geriatr. Psychiatry. 16(5), ,2008,343–354 Qiu C, Winblad B, Fratiglioni L. The age-dependent relation of blood pressure to cognitive function and dementia. Lancet Neurol. 4(8), 2005, 487–499 Luchsinger JA, Gustafson DR. Adiposity, Type 2 diabetes, and Alzheimer’s disease. J. Alzheimers Dis. 16(4), 2009, 693–704 Solfrizzi V, Frisardi V, Capurso C et al. Dietary fatty acids in dementia and predementia syndromes: epidemiological evidence and possible underlying mechanisms. Ageing Res. Rev. 9(2), 2010,184–199 Parrott MD, Greenwood CE. Dietary influences on cognitive function with aging. From high-fat diets to healthful eating. Ann. NY Acad. Sci. 1114, 2007, 389–397 Luchsinger JA, Noble JM, Scarmeas N. Diet and Alzheimer’s disease. Curr. Neurol. Neurosci. Rep. 7(5), 2007,366–372 Peters R, Peters J, Warner J, Beckett N, Bulpitt C. Alcohol, dementia and cognitive decline in the elderly. a systematic review. Age Ageing 37(5), 2008,505–512 Frisardi V, Solfrizzi V, Seripa D et al. Metabolic-cognitive syndrome: a cross-talk between metabolic syndrome and Alzheimer’s disease. Ageing Res. Rev. 9(4), 2010,399–417 O´Keefe J.H; Gheewala N.M, O´Keefe J.O., Dietary Strategies for Improving Post-Prandial Glucose, Lipids, Inflammation, and Cardiovascular Health, J Am Coll Cardiol. 2008;51-3 Hulea St, Vitamins Minerals and Oxidative Stress, Ed. SUniversal-Publishers, Boca Raton Florida USA, 2008 Mellen P, Cefalu W, Herrington D. Diabetes, the metabolic syndrome, and angiographic progression of coronary artery disease in postmenopausal women. Arterioscler Thromb Vasc Biol 2006, 26: 189–93. O´Keefe J, Bell D. The post-prandial hyperglycemia/hyperlipemia hypothesis: a hidden cardiovascular risk factor? Am J Cardiol 2007; 100:899 –904 O’Riordan M. Fish Oil Added to Statin Therapy Reduces Risk For Major Coronary Events. Lancet 2007;369:1062-1063, 1090-1098 Scarmeas N, Stern Y, Tang MX, Mayeux R, Luchsinger JA. Mediterranean diet and risk for Alzheimer’s disease. Ann. Neurol. 59(6), 2006, 912–921 O´Keefe H.J, Gheewala N.M, Joan O´Keefe, Dietary Strategies for Improving Post-Prandial Glucose, Lipids, Inflammation, and Cardiovascular Health , J. Am. Coll. Cardiology, 2008, 51(3) Stevenson E, Williams C, Mash L, Phillips B, et al. Influence of high-carbohydrate mixed meals with different glycemic indexes on substrate utilization during subsequent exercise in women. Am J Nutr 2006; 84:354–60. Byrans JA, Judd PA, Ellis PR. The effect of consuming instant black tea on postprandial plasma glucose and insulin concentrations in healthy humans. J Am Coll Nutr 2007;26:471–7 Solfrizzi V, D’Introno A, Colacicco AM et al. Dietary fatty acids intake: possible role in cognitive decline and dementia. Exp. Gerontol. 40(4), 257–270 (2005) Assisi A, Banzi R, Buonocore C et al. Fish oil and mental health. the role of n-3 long-chain polyunsaturated fatty acids in cognitive development and neurological disorders. Int. Clin. Psychpharmacol. 21(6), 319–336 (2006). Cunnane SC, Plourde M, Pifferi F, et al. Fish, docosahexaenoic acid and Alzheimer’s disease. Prog. Lipid Res. 48(5), 239–256 (2009). Solfrizzi V, Frisardi V, Capurso C et al. Dietary fatty acids in dementia and predementia syndromes: epidemiological evidence and possible underlying mechanisms. Ageing Res. Rev. 9(2), 184–199 (2010). Dai Q, Borenstein A, Wu Y, Jackson JC, Larson EB. Fruit and vegetable juices and Alzheimer’s disease. the Kame project. Am. J. Med. 119(9), 751–759 (2006). Parrott MD, Greenwood CE. Dietary influences on cognitive function with aging. From high-fat diets to healthful eating. Ann. NY Acad. Sci. 1114, 389–397 (2007). Zorila Corina, Natural antioxidants today, in vol. Environmental Biodiversity and Health, proceeding of Euroregional Conference, May 11-13, 2007 Arad, Ed.“V. Goldis University Press,”p. 236-238, Bjelakovic G., Nikolova D., Gluud L., at al, Mortality in Randomized Trials of Antioxidant Supplements for Primary and Secondary Prevention: Systematic Review and Meta-Analysis, Medscape Family Medicine,Posted 03/27/2007 Solfrizzi V.,s Panza F., Frisardi V., et al, Diet and Alzheimer’s Disease Risk Factors or Prevention, Expert Rev Neurother.2011; 11(5):677-708
*Correspondence: